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Exercise-induced homeostatic perturbations provoked by singles tennis match play with reference to development of fatigue

¹ Faculty of Physical Activity and Sport Sciences, University of Alcala, Madrid, Spain
² Faculty of Physical Activity and Sport Sciences, University of Leon, Spain
³ Team Sport Research Group, Facoltà di Scienze Motorie, Università di Verona, Italy

Correspondence to:
Alberto Mendez-Villanueva, Facultad de Ciencias de la Actividad Física y el Deporte, Universidad de Alcalá, Campus Universitário, Ctra. Madrid-Barcelona Km. 3,600, 28871 Alcalá de Henares (Madrid), Spain; amendezvillanueva{at}yahoo.com

This review addresses metabolic, neural, mechanical and thermal alterations during tennis match play with special focus on associations with fatigue. Several studies have provided a link between fatigue and the impairment of tennis skills proficiency. A tennis player’s ability to maintain skilled on-court performance and/or optimal muscle function during a demanding match can be compromised as a result of several homeostatic perturbations, for example hypoglycaemia, muscle damage and hyperthermia. Accordingly, an important physiological requirement to succeed at competitive level might be the player’s ability to resist fatigue. However, research evidence on this topic is limited and it is unclear to what extent players experience fatigue during high-level tennis match play and what the physiological mechanisms are that are likely to contribute to the deterioration in performance.

Abbreviations: ATP, adenosine triphosphate; CHO, carbohydrate; PCr, phosphocreatine; RFD, rate of force development; SSC, stretch–shortening cycle

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Commentary on ""

Tim Wood
Br. J. Sports Med. 2007 41: 722. [Extract] [Full Text] [PDF]